Publications

Oh S, Geistlinger L, Ramos M, Blankenberg D, van den Beek M, Taroni JN, et al. GenomicSuperSignature facilitates interpretation of RNA-seq experiments through robust, efficient comparison to public databases. Nature Commun. 2022;13: 3695. [PDF]

Asnicar F, Berry SE, Valdes AM, Nguyen LH, Piccinno G, Drew DA, et al. Microbiome connections with host metabolism and habitual diet from 1,098 deeply phenotyped individuals. Nat Med. 2021;27: 321–332.

Oh S, Abdelnabi J, Al-Dulaimi R, Aggarwal A, Ramos M, Davis S, et al. HGNChelper: identification and correction of invalid gene symbols for human and mouse. F1000Res. 2020;9: 1493.

Geistlinger L, Oh S, Ramos M, Schiffer L, LaRue RS, Henzler CM, et al. Multiomic Analysis of Subtype Evolution and Heterogeneity in High-Grade Serous Ovarian Carcinoma. Cancer Res. 2020;80: 4335–4345.

Oh S, Geistlinger L, Ramos M, Morgan M, Waldron L, Riester M. Reliable Analysis of Clinical Tumor-Only Whole-Exome Sequencing Data. JCO Clinical Cancer Informatics. 2020. pp. 321–335. doi:10.1200/cci.19.00130

Ramos M, Geistlinger L, Oh S, Schiffer L, Azhar R, Kodali H, et al. Multiomic Integration of Public Oncology Databases in Bioconductor. JCO Clin Cancer Inform. 2020;4: 958–971.

Carey VJ, Ramos M, Stubbs BJ, Gopaulakrishnan S, Oh S, Turaga N, et al. Global Alliance for Genomics and Health Meets Bioconductor: Toward Reproducible and Agile Cancer Genomics at Cloud Scale. JCO Clin Cancer Inform. 2020;4: 472–479.

Camacho-Rivera M, Rice SL, Oh S, Paris M, Akpara E, Molina J, et al. Cancer Health Impact Program (CHIP): Identifying Social and Demographic Associations of mHealth Access and Cancer Screening Behaviors Among Brooklyn, New York, Residents. Cancer Epidemiol Biomarkers Prev. 2019;28: 478–485.

Ghezraoui H, Piganeau M, Renouf B, Renaud B, Sallmyr A, Ruis B, Oh S, et al. Chromosomal translocations in human cells are generated by canonical nonhomologous end-joining. Mol Cell. 2014;55: 829–842.

Jones RE, Oh S, Grimstead JW, Zimbric J, Roger L, Heppel NH, et al. Escape from telomere-driven crisis is DNA ligase III dependent. Cell Rep. 2014;8: 1063–1076.

Waters CA, Strande NT, Pryor JM, Strom CN, Mieczkowski P, Burkhalter MD, Oh S, et al. The fidelity of the ligation step determines how ends are resolved during nonhomologous end joining. Nat Commun. 2014;5: 1–11.

Oh S, Harvey A, Zimbric J, Wang Y, Nguyen T, Jackson PJ, et al. DNA ligase III and DNA ligase IV carry out genetically distinct forms of end joining in human somatic cells. DNA Repair. 2014;21: 97–110.

Smith S, Fox J, Mejia M, Ruangpradit W, Saberi A, Kim S, Choi Y, Oh S, et al. Histone deacetylase inhibitors selectively target homology dependent DNA repair defective cells and elevate non-homologous end joining activity. PLoS One. 2014;9: e87203.

Yang Y, Durando M, Smith-Roe SL, Sproul C, Greenwalt AM, Kaufmann W, Oh S, et al. Cell cycle stage-specific roles of Rad18 in tolerance and repair of oxidative DNA damage. Nucleic Acids Res. 2013;41: 2296–2312.

Oh S, Wang Y, Zimbric J, Hendrickson EA. Human LIGIV is synthetically lethal with the loss of Rad54B-dependent recombination and is required for certain chromosome fusion events induced by telomere dysfunction. Nucleic Acids Res. 2013;41: 1734–1749.

Strande N, Roberts SA, Oh S, Hendrickson EA, Ramsden DA. Specificity of the dRP/AP lyase of Ku promotes nonhomologous end joining (NHEJ) fidelity at damaged ends. J Biol Chem. 2012;287: 13686–13693.

Fattah FJ, Lichter NF, Fattah KR, Oh S, Hendrickson EA. Ku70, an essential gene, modulates the frequency of rAAV-mediated gene targeting in human somatic cells. Proc Natl Acad Sci U S A. 2008;105: 8703–8708.